Diary of Unknown Symptoms

Mystery of the Internal Vibration

Entry for August 10, 2007

When I discovered vitamin and mineral deficiencies for the very first time, I have always said that there was a problem with the myelin sheaths based on how I felt with these weird feelings in my head and with the hydro transmission towers. Of course everyone thought I was crazy but I knew I had a problem…I just didn’t understand how or why?

Came across this today which is only a “hypothesis” but it makes perfect sense to me. It really makes me wonder where I would be now if I didn’t take the bull by the horns to figure out my health issues myself.

Accumulated copper and low vitamin B2 can exacerbate low uric acid levels, which in turn is hypothesized to lead to myelin degeneration seen in Multiple Sclerosis.

Low uric acid levels may be associated with a molybdenum deficiency, copper toxicity, and a worsening of multiple sclerosis.

August 12, 2007 Posted by | Health | , , , , , | Leave a comment

Entry for July 14, 2007

Just when you think you’ve read everything you need to know…

“Phosphorus is vital to collagen production”

“If you don’t have enough phosphorus, you can’t make lecithin”

Now I’ve tried Lecithin a long time ago as it was recommended to me and I didn’t notice any difference. Maybe it wasn’t being absorbed properly because of the low phosphorus?? It’s certainly possible and I think I still have some left over…I’ll have to see if I can find it again… It’s not considered a vitamin or a mineral so it could be overlooked so easily. And maybe it was…

Deficiency Diseases and Good Nutrition: Phosphorus

Phosphorus has more functions than any other mineral in your body. Eighty to eighty five percent of all the phosphorus in your body is in your bones and teeth in the form of calcium phosphate. The 1 1/2 – 2 pounds of phosphorus in your body helps to keep your bones and teeth strong. Phosphorus is important to your bones for another reason. Phosphorus is vital to collagen production and bone is 3/4 collagen. Tendons, ligaments, cartilage, skin, and eyes, are composed greatly of collagen. Without phosphorus, your body could not make any of the above vital connective tissues and organs.

Phosphorus joins up with fatty acids in the body to form phospholipids. Phospholipids have a very interesting characteristic: they are soluble in water and fat. The fatty acid makes phospholipids soluble in fat and the phosphorus makes it soluble in water. This unique trait makes phospholipids very useful as the main component in cell membranes. This allows for fat and water soluble nutrients to pass in and out of the cell. Each cell needs fat and water soluble nutrients to live, so this property is vital. Cell membranes would collapse without phospholipids.

Phospholipids also have a job in the blood. Phospholipids keep blood fat in small globules. Large globules can get stuck on artery walls, which can eventually cause heart disease. Lecithin is a phospholipid used in chocolate to keep it smooth. In the body, phospholipids keep the blood smooth.

Lecithin helps to make bile at an even rate. Bile helps you emulsify fats (break into very small globules). If you don’t have enough phosphorus, you can’t make lecithin to help make bile, which results in painful gallstones. Lecithin helps to keep fat from accumulating in the liver (cirrhosis).

Phosphorus helps to transport fatty acids throughout your body. It maintains the proper pH in blood and stomach so the needed reactions can take place. Phosphorus has to be present to use fat soluble vitamins A, D, E, and K. Phosphorus combines with carbohydrates to be stored as energy. Phosphorus is a MAIN component in ATP (adenosine triphosphate), which is the primary source of energy for all living cells. Without ATP, you could not catalyze any reaction.

Phosphorus is needed in myelin, which is the fatty covering on nerves to help the impulses travel faster. Phosphorus fatty acid compounds make up 1/3 of the brain’s dry weight. Muscles could not contract without phosphorus because the nerves could not properly transmit the impulse without myelin. The muscle would not have the energy to contract without phosphorus containing ATP.

July 14, 2007 Posted by | Health | , , , , | Leave a comment

Entry for March 25, 2007


Benefits & Deficiency Symptoms

Sulfur is known as a healing mineral. Sulfur aids every cell in the elimination of toxic substances through agitation. Sulfur aids in enzyme reactions and protein synthesis. Sulfur also plays an important role in cellular respiration. Sulfur is the flexible bond that connects cells; Sulfur is the lubricant found between joints. A deficiency of water-soluble Sulfur can lead to a variety of conditions ranging from skin irritations and rashes to total breakdown of cellular regeneration. Pain and inflammation associated with various muscle and skeletal disorders indicate a deficiency of Sulfur.

It is believed that Sulfur can repair the myelin sheath, the protector on the end of every nerve in the body. Damage to the myelin sheath causes the shaking condition in palsy, Multiple Sclerosis, Lorenzo’s Disease and many other disease conditions where motor functions are uncontrollable.

Chronic or severe allergies to materials such as dust, pollen, wool, animal hair, feathers, etc. (with symptoms ranging from respiratory congestion to inflammation, itching and general discomfort) can be relieved with Sulfur. It increases blood circulation, reduces back pain, and relieves migraine headaches. Sulfur promotes muscle healing. Sulfur scavenges free radicals, beautifies the skin, and relieves allergies to food. Sulfur also controls acidity in stomach ulcers. Sulfur is important for carbohydrate metabolism and speeds wound healing. Sulfur is stored in the brain, nerves, bowel, liver and in all body cells, especially in the skin, hair and nails.

March 25, 2007 Posted by | Health | , | Leave a comment

Entry for March 22, 2007

Copper and the importance on the Nervous System… has a role with the neurotransmitters and can effect serotonin and the myelin sheath. All things that I have suspected for a long time.


Copper (Cu) is an essential trace element for humans and animals. In the body, copper shifts between the cuprous (Cu1+) and the cupric (Cu2+) forms, though the majority of the body’s copper is in the Cu2+ form. The ability of copper to easily accept and donate electrons explains its important role in oxidation-reduction (redox) reactions and the scavenging of free radicals. Although Hippocrates is said to have prescribed copper compounds to treat diseases as early as 400 B.C., scientists are still uncovering new information regarding the functions of copper in the human body

Central Nervous System

A number of reactions essential to normal function of the brain and nervous system are catalyzed by cuproenzymes. Neurotransmitter synthesis: Dopamine-b-monooxygenase catalyzes the conversion of dopamine to the neurotransmitter norepinephrine.

Metabolism of neurotransmitters: Monoamine oxidase (MAO) plays a role in the metabolism of the neurotransmitters norepinephrine, epinephrine, and dopamine. MAO also functions in the degradation of the neurotransmitter serotonin, which is the basis for the use of MAO inhibitors as antidepressants.

Formation and maintenance of myelin: The myelin sheath is made of phospholipids whose synthesis depends on cytochrome c oxidase activity.

March 22, 2007 Posted by | Health | , , , | Leave a comment

Entry for February 15, 2007

Same as yesterday: No vibration last night or this morning. There is definitely a difference and I’m beginning to think it has to do with the additional copper intake. I started to wonder if this was the reason for my weak knees? A copper deficiency effected my ligaments? I need to do more research and I come across a link between copper, candida, stressed adrenals, neurotransmitters, serotonin production, thyroid hormones, myelin sheaths and??? Mitral Valve Prolapse!

My jaw almost dropped to the ground…


Copper is the body ís natural anti-candida agent. Farmers often spray copper sulfate on fruits and vegetables to kill yeast and molds. Water departments may add copper compounds to drinking water for the same reason. Copper may be added to swimming pools and hot tubs to control yeast. Copper favors aerobic metabolism which disables yeast.

Candida Albicans

Everyone with candida has a copper imbalance. When copper is out of balance, our bodies cannot control yeast overgrowth. This often lead to chronic candida albicans infections that are resistant to treatment.

Adrenal Glands

Most often, copper is present but not available to the body. This is due to deficiency of ceruloplasmin, a copper binding protein. Adrenal hormones are required to produce ceruloplasmin. Underactive, exhausted adrenal glands or sluggish liver activity cause a decrease in ceruloplasmin production. As a result, copper is not properly bound and is less available to the body. Until the copper-adrenal-liver condition is corrected, candida is difficult to control.

Birth control pills impair copper metabolism. Some antibiotics act by removing copper from the liver. Steroid therapy slows the adrenal glands which makes copper less available. All can cause or aggravate candida.


Copper is critical for energy production in the cells. It is also involved in nerve conduction, connective tissue, the cardiovascular system and the immune system. Copper is closely related to estrogen metabolism, and is required for women’s fertility and to maintain pregnancy. Copper stimulates production of the neurotransmitters epinephrine, norepinephrine and dopamine. It is also required for monoamine oxidase, an enzyme related to serotonin production.

Copper is also important for the production of the thyroid hormone called thyroxine and is necessary for the synthesis of phospholipids found in myelin sheaths that cover and protect nerves.


Physical conditions associated with copper imbalance include arthritis, fatigue, adrenal burnout, insomnia, scoliosis, osteoporosis, heart disease, cancer, migraine headaches, seizures, fungal and bacterial infections including yeast infection, gum disease, tooth decay, skin and hair problems and female organ conditions including uterine fibroids, endometriosis and others. Mental and emotional disorders related to copper imbalance include spaciness, depression, mood swings, fears, anxiety, phobias, panic attacks, violence, autism, schizophrenia, and attention deficit disorder. Copper deficiency is associated with aneurysms, gout, anemia and osteoporosis.


Copper is required for collagen formation. Copper deficiency is association with atherosclerosis and other cardiovascular conditions. Excess copper or biounavailable copper often cause connective tissue problems, interfering with the disulfide bonds in connective tissue. Symptoms may include stretch marks, tendon and ligament weakness, mitral valve prolapse, skin and hair problems and other conditions affecting connective tissue.

February 15, 2007 Posted by | Health | , , , , , | Leave a comment

Entry for September 03, 2006


A while back I had mentioned to my mother about having a B12 deficiency because she is a vegetarian. Her symptom is that she has sometimes has a nerve tingling on the top of her head. It’s the same kind of tingling that I have so I suspect that it’s related to the myelin sheath. She doesn’t seem to think it a vitamin deficiency so I did a bit of research and I came across this:


B12 is an essential part of maintaining the fatty acid balance in the myelin sheath, which is the living insulation cable around our nerves.  Cholesterol is an essential part of the myelin sheath which is 80% phospholipids and 20% proteins.  Essential fatty acids EPA and DHA which are found in fish oils concentrate in myelin.  B6 is needed for its development and growth.  Taurine stabilises electrical activity.  Carnitine inhibits degeneration of the sheath and thickens the fibres improving nerve conduction, particularly if diabetes is present.

Low B12 levels also contribute to carnitine loss through the kidneys.  So again where carnitine retention is essential for polio survivors, ensuring good levels of B12 are very important.

The simple test is to look at the moons on your fingernails.  Good B12 levels will give you white moon that come a third of the way up the thumb-nails.  We should have moons on all fingers, thumbs are the last to go.  If in doubt, ask your doctor to do a blood test for B12.  While he is at it ask for folic acid & thyroid function test (include T3, T4 as well as TSH) because low thyroid levels will increase your cholesterol levels as well as contributing to fatigue. When thyroid levels are low we feel the cold.

My parents came down today for a visit so I asked my mother if I could look at her fingernails. She had very little or no white moons in her fingernails so I asked if I could take a picture.

September 3, 2006 Posted by | Health | , | Leave a comment

Entry for August 29, 2006

From the prevous entry:

I ask him if there is any kind of test that could measure any kind of demyelination and he answered “No not really…”

Here’s what I found:

Nerve Conduction Velocity (NCV) Test

What is it?
A nerve conduction velocity test, also called a nerve conduction study, measures how quickly electrical impulses move along a nerve. It is often done at the same time as an electromyogram, in order to exclude or detect muscle disorders.

A healthy nerve conducts signals with greater speed and strength than a damaged nerve. The speed of nerve conduction is influenced by the myelin sheath the insulating coating that surrounds the nerve.

Most neuropathies are caused by damage to the nerve’s axon rather than damage to the myelin sheath surrounding the nerve. The nerve conduction velocity test is used to distinguish between true nerve disorders (such as Charcot-Marie-Tooth disease) and conditions where muscles are affected by nerve injury (such as carpal tunnel syndrome).

Why should I do it?
This test is used to diagnose nerve damage or dysfunction and confirm a particular diagnosis. It can usually differentiate injury to the nerve fiber (axon) from injury to the myelin sheath surrounding the nerve, which is useful in diagnostic and therapeutic strategies.

How is it performed?
During the test, flat electrodes are placed on the skin at intervals over the nerve that is being examined. A low intensity electric current is introduced to stimulate the nerves.

The velocity at which the resulting electric impulses are transmitted through the nerves is determined when images of the impulses are projected on an oscilloscope or computer screen. If a response is much slower than normal, damage to the myelin sheath is implied. If the nerve’s response to stimulation by the current is decreased but with a relatively normal speed of conduction, damage to the nerve axon is implied.

And perhaps the best of all…I found this about Benfotmaine. The supplement that the Neurologist had never heard of:

Shielding Nerve Structure

While most anti-AGE supplements rely on test-tube browning experiments as the evidence of efficacy, Benfotiamine has been proven in multiple real-world human and animal studies to reduce AGE formation and support tissue structure and function in diabetics.

Most impressively, many randomized, double-blind, placebo-controlled human trials have proven that Benfotiamine powerfully supports nerve function in diabetic neuropathy. In one trial, 24 people suffering with diabetic neuropathy took either Benfotiamine (plus doses of common B6 and B12 similar to those used in mutivitamins) or a look-alike dummy pill, spread out into three pills over the course of the day, for twelve weeks. The participants started with 320 milligrams of Benfotiamine per day for the first two weeks, followed by 120 milligrams for the rest of the trial. Before and after the trial, the function of patients nerve cells were tested using nerve conduction velocity (NCV) and vibratory perception threshold (which tests the nerves sensitivity by determining the lowest level at which vibrations applied at key nerve sites are first felt).

At the end of the trial, the vibration perception threshold had clearly improved by 30% in those who had taken the Benfotiamine supplements, while it had worsened in the placebo group by 5% at one site and by 32% at another. At the same time, people taking Benfotiamine experienced statistically significant improvements in nerve conduction velocity from the feet, even as this aspect of nerve function deteriorated in those taking the look-alike pills.

This is exactly why I don’t like doctors anymore.

August 29, 2006 Posted by | Health | , , , | Leave a comment

Entry for August 17, 2006

Vaccine Induced Demyelination

Myelination is an essential part of human brain development. Nerves can only conduct pulses of energy efficiently if covered by myelin. Like insulation on an electric wire, the fatty coating of myelin keeps the pulses confined and maintains the integrity of the electrical signal so that it has a high signal-to-noise ratio. When the insulation on a wire is damaged or destroyed, the flow of electrical current may be interrupted and a short-circuit occurs.

Oligodendrocyte cells give white matter its color by manufacturing myelin. If myelin falls into disrepair, nerve axons cease to function, even though they themselves aren’t damaged. Protecting oligodendrocytes after brain or spinal cord injury might keep nerve cells intact.

At birth, relatively few pathways have myelin insulation. Myelination in the human brain continues from before birth until at least 20 years of age. Up until the age of 10 or so, vast areas of the cortex are not yet myelinated. Up to the age of 20, large areas of the frontal lobes are not yet myelinated.

Myelination begins in the developmentally oldest parts of the brain, like the brain stem, moving to the areas of the nervous system that have developed more recently, like the prefrontal lobe and cortex. Myelin spreads throughout the nervous system in stages, which vary slightly in each individual. Impairment of myelination can alter neural communication without necessarily causing severe CNS (central nervous system) damage.

The prefrontal portions of the cerebrum have a profound influence on human behavior. If an individual is injected with vaccines, most of which have adjuvants like mercury and aluminum compounds, as well as foreign proteins (some from other species in which the vaccines were grown) and biological organisms, unprotected nerves may be impacted. The argument for a role of vaccines in the development of autistic disorders hinges on these biological effects upon nerves, damaging them in a way that influences behavior and learning patterns.

August 17, 2006 Posted by | Health | , , , | Leave a comment

Entry for August 16, 2006

I was talking to my mother the other day about my recent discoveries and I was explaining about tingling in my head and how it was related to the myelin sheath. She told me that she has felt the same kind of tingling in her head and often wondered if it was the same as mine. She says it’s very mild and doesn’t happen all of the time. My mother is a vegetarian and has been for most of her life so there’s no way that it could be related to riboflavin and magnesium deficiencies.

I grab my newest book “Encyclopedia of Nutritional Supplements” and I take it into work. It’s a long commute so it’s perfect reading. I look up myelin sheath in the rear index and to my surprise there is a listing for page 127.

It talks about the role of vitamin B12 in the development of the myelin sheath and I already knew that. But it goes on to say that vegetarians often need B12 supplements as significant quantities are found in animal foods.

I think my mother has a B12 deficiency so I call her up and tell her to see her doctor for a blood test and she agrees. She says that she has taken B12 from time to time but never noticed any difference. Doctor Google?

Does this affect all vegetarians?

Reports from around the world reveal that many long-term total vegetarians (vegetarians who do not use any eggs, meat, fish, poultry or dairy products) are especially at risk of vitamin B12 deficiency. Unfortunately, many total vegetarians fail to recognize the seriousness of B12 deficiency. Total vegetarians often have low serum B12 levels and may manifest neuropsychiatric disorders. While oral B12 supplements can restore serum levels of B12 and eliminate macrocytic anemia, the neurological disorders may persist even months after treatment. In some cases the damage done to the nervous system is not reversible.

On rare occasions a lacto-ovo-vegetarian (one that uses dairy products and eggs, but no meat, fish or poultry) may also have a low serum B12 level if their intake of vitamin B12 containing foods is very low. Most of those with low serum B12 levels can correct the macrocytic anemia with oral B12 supplements or an injection of B12. In one study, the serum B12 levels of adult lacto-ovo-vegetarians dropped 35 percent only two months after switching to a total vegetarian diet. This rapid drop may be the result of low B12 stores in the liver. It should be emphasized, however, that vitamin B12 deficiency most often occurs in total vegetarians.

Vitamin B12 also maintains the fatty sheath, called myelin, that surrounds and protects nerve fibers and promotes their normal growth. Like insulation around copper wires, this sheath allows your radiating network of nerves to send their electrical messages without short-circuiting. When B12 is missing, the myelin sheath breaks down, which eventually leads to nerve damage.

August 16, 2006 Posted by | Health | , , , | Leave a comment

Entry for August 15, 2006



The principle function of oligodendrocytes is to provide support to axons and to produce the Myelin sheath, which insulates axons.  Myelin is 80% lipid and 20% protein and allows for the efficient conduction of action potentials down the axon.  Oligodendrocytes unlike Schwann cells of the PNS, form segments of myelin sheaths of numerous neurons at once.  As can be seen in the above illustration, the processes of a given oligodendrocyte wrap themselves around portions of the surrounding axons.  As each process wraps itself around, it forms layers of myelin. Each process thus becomes a segment of the axon’s myelin sheath.

August 15, 2006 Posted by | Health | , | Leave a comment

Entry for August 14, 2006

I start the day organizing the myelin sheath building vitamins. B1, B6, B12, folate, vitamin C, D, and E, Magnesium, Omega 3 and Lecithin.

Here’s what I find on magnesium absorption:

Best taken: At bedtime with calcium in the ratio of one or two parts calcium to one part magnesium. Most absorbable forms are magnesium orotate and amino-chelated magnesium. Magnesium gluconate and magnesium chloride are less effective but can be taken orally.

The process of digestion and absorption of magnesium is very similar to that of calcium. The suggested ratio of intake of these two vital nutrients is about 2:1, calcium to magnesium. Magnesium also requires an acidic stomach environment for best absorption, so taking it between meals or at bedtime is recommended. Meals high in protein or fat, a diet high in phosphorus or calcium (calcium and magnesium can compete), or alcohol use may decrease magnesium absorption.

Usually, about 40-50 percent of the magnesium we consume is absorbed, though this may vary from 25-75 percent depending on stomach acid levels, body needs, and dietary habits.

The levels of magnesium used by physicians are commonly in the range of 600-1,000 mg.; however, the researchers in the kidney stone studies used only 200-300 mg. of supplemental magnesium oxide. Calcium and magnesium are both alkaline minerals, so they are not taken with or after meals, as they can reduce stomach acid as well as being absorbed poorly when taken with food. They are absorbed better when taken between meals or on an empty stomach, especially with a little vitamin C as ascorbic acid.

Many calcium-magnesium combinations are formulated with hydrochloric acid and vitamin D to aid the mineral absorption. And taking them before bedtime may be very helpful in increasing utilization of both these important minerals and lead to a sleep-filled night.

The best forms of supplemental magnesium seem to be the ones chelated to an amino acid (magnesium glycinate, magnesium taurate) or a krebs cycle intermediate (magnesium malate, magnesium citrate, magnesium fumarate). These forms seem to be better utilized, absorbed, and assimilated. Try to stay away from inorganic forms of magnesium like magnesium chloride or magnesium carbonate because they may not be absorbed as well and may cause gastric disturbances.

The magnitude of increased absorption with true amino acid chelates is biologically profound. Comparing a magnesium amino acid chelate to magnesium oxide (a typical salt found in many supplements) the amino acid chelate had 9 times the absorption as the oxide. This study compared the use of the single mineral. If another inorganic mineral were given at the same time, such as calcium carbonate, the usual effect would be no reduction of the amino acid chelate, but a further 70-80% reduction of magnesium absorption from the oxide form. The net effect on absorption of chelated to inorganic magnesium, if two or more inorganic minerals are taken, can be 20-30 fold greater absorption or more of the amino acid chelate.

Magnesium-containing products are either used in treating a primary or secondary magnesium deficiency or are given for prophylaxis as food supplement. Various inorganic (oxides, chlorides, sulfates and carbonates) and organic (citrates, aspartates) magnesium compounds are available for oral administration.

A comparison of the magnesium content of food as stated in tables of nutritional values and the actual analytical values showed that food contains less magnesium than reported in the literature. This means that a deficiency may arise even when the calculated intake is sufficient. Also, food processing, such as polishing rice or cooking fresh vegetables, can lead to loss of magnesium. However, insufficient supply due to nutrition is only one of many causes of magnesium deficiency.

Those suffering from chronic fatigue syndrome also seem to do better with magnesium given by injection. This may be due to the superior absorption of injectable magnesium or because high concentrations in the body are necessary for maximal therapeutic effects.

August 14, 2006 Posted by | Health | , , , , , , , | Leave a comment

Entry for August 14, 2006

And this:

It is now known the cause for the destruction of the myelin in the lesions is overactivation of the microglia in the region of the myelin. An enzyme that converts glutamine to glutamate called glutaminase increases tremendously, thereby greatly increasing excitotoxicity. Mercury also activates microglia, even in subtoxic doses.
Any dietary excitotoxin can activate the microglia, thereby greatly aggravating the injury. This includes the aspartate in aspartame. The methanol adds to this toxicity as well. Now, the secret to treatment appears to be shutting down, or at least calming down, the microglia. It has been found that the antibiotic minocycline powerfully shuts down the microglia. I tried this treatment on a friend of mine who just came down with fulmanant MS. He was confined to a wheelchair. I had him placed on minocycline and now, just a few weeks later, he is walking.
The good news is that other things also calm the microglia-the most potent are: silymarin, curcumin and ibuprophen. Phosphatidylcholine helps re-myelinate the nerve sheaths that are damaged, as does B12, B6, B1, vitamin D, folate, vitamin C, natural vitamin E (mixed tocopherols) and L-carnitine. DHA plays a major role in repairing the myelin sheath. Vitamin D may even prevent MS, but it acts as an immune modulator, preventing further damage – the dose is 2000 IU a day. Magnesium, as magnesium malate, is needed in a dose of 500 mg 2X a day. They must avoid all excitotoxins, even natural ones in foods-such as soy, red meats, nuts, mushrooms and tomatoes. Avoid all fluoride and especially all vaccinations since these either inhibit antioxidant enzymes or triggers harmful immune reactions.

Phosphatidylcholine helps re-myelinate the nerve sheaths. What is this stuff and how can I get me some?

Phosphatidylcholine is a phospholipid that is a major constituent of cell membranes. Phosphatidylcholine is also known as 1, 2-diacyl-:ussn:ue-glycero-3-phosphocholine, PtdCho and lecithin.

So Phosphatidylcholine, is a fancy name for lecithin. Perfect! I already have some. Interestingly enough, three of the main supplements that are required for rebuilding the myelin sheath, the iridologist told me I didn’t need them:

Magnesium, Vitamin D and DHA from Omega 3.

More about Magnesium Malate:

Heavy Metal Toxicity

Magnesium malate may be one of the few compounds capable of pulling aluminum from the body over time.

And my hair analysis came up with high aluminum levels and this form of magnesium is recommened for building the myelin sheaths and removing aluminum from the body. The Naturopath didn’t mention this? She gave me a standard calcium/magnesium but I’m sure it wasn’t magnesium malate.

August 14, 2006 Posted by | Health | , , , , , , , , | Leave a comment

Entry for August 14, 2006

More googling turned up this:

Ultrastructure of the optic nerve in magnesium-deficient rats.

Department of Ophthalmology and Visual Sciences, Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan.

PURPOSE: The ultrastructure of the optic nerves in magnesium (Mg)-deficient rats was studied to elucidate the function of Mg.

METHODS: After delivery, mother Wistar Kyoto rats were fed a low-Mg diet containing 0.1 mg Mg/100 g diet with all other nutrients and distilled and deionized water. Infant rats were suckled by their mothers for 21 days and then fed the same Mg-deficient diet. Control mother rats were fed commercial rat pellets containing 24 mg Mg/100 g diet and all other nutrients. The optic nerves were examined by electron microscopy at 6 weeks of age.

RESULTS: In the Mg-deficient rats, serum Mg levels were significantly lower and calcium levels higher than in the control rats. Ultrastructural findings were: significantly fewer myelinated axons and significantly thinner myelin sheaths in the Mg-deficient rats than in the control and pair-fed controls, and more numerous unmyelinated axons. There were multifocal areas of destruction and necrosis in the optic nerve of Mg-deficient rats.

CONCLUSIONS: This study suggests that the optic nerve needs Mg for the development and maintenance of its cell structure.

Copyright 2003 S. Karger AG, Basel

August 14, 2006 Posted by | Health | , | Leave a comment


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